Reading
Everyone: Flake, ch. 16
CS594: Bar-Yam, Sections 7.1, 7.2.1-7.2.2 (pp. 621-48)

Universal Properties
What leads to these expanding rings and spirals in very different systems?
Under what conditions do these structures form?
What causes the rotation?
These are all examples of excitable media

Excitable Media
Examples of Excitable Media
Slime mold amoebas
Cardiac tissue (& other muscle tissue)
Cortical tissue
Certain chemical systems (e.g., BZ reaction)
Hodgepodge machine

Characteristics of
Excitable Media
Local spread of excitation
for signal propagation
Refractory period
for unidirectional propagation
Decay of signal
avoid saturation of medium

Behavior of Excitable Media
Stimulation
Relay (Spreading Excitation)
Continued Spreading
Recovery
Restimulation
Typical Equations for
Excitable Medium
(ignoring diffusion)
Excitation variable:
Recovery variable:

Nullclines
Rest State
Stability
Super-threshold Excitation
Phase 1: Increasing Excitation
Phase 2: Start of Extinction
Phase 3: End of Extinction
Phase 4: Recovery
Elevated Thresholds During Recovery
Modified Martiel & Goldbeter Model for Dicty Signalling
Variables (functions of x, y, t):
b = intracellular concentration of cAMP
g = extracellular concentration of cAMP
= fraction of receptors in active state

Equations
Positive Feedback Loop
Extracellular cAMP increases
(g increases)
Þ Rate of synthesis of intracellular cAMP increases
(F increases)
Þ Intracellular cAMP increases
(b increases)
Þ Rate of secretion of cAMP increases
(Þ Extracellular cAMP increases)

Negative Feedback Loop
Extracellular cAMP increases
(g increases)
Þ cAMP receptors desensitize
(f1 increases, f2 decreases, r decreases)
Þ Rate of synthesis of intracellular cAMP decreases
(F decreases)
Þ Intracellular cAMP decreases
(b decreases)
Þ Rate of secretion of cAMP decreases
Þ Extracellular cAMP decreases
(g decreases)

Dynamics of Model
Unperturbed
Þ cAMP concentration reaches steady state
Small perturbation in extracellular cAMP
Þ returns to steady state
Perturbation > threshold
Þ large transient in cAMP,
 then return to steady state
Or oscillation (depending on model parameters)

Circular & Spiral Waves Observed in:
Slime mold aggregation
Chemical systems (e.g., BZ reaction)
Neural tissue
Retina of the eye
Heart muscle
Intracellular calcium flows
Mitochondrial activity in oocytes

Cause of
Concentric Circular Waves
Excitability is not enough
But at certain developmental stages, cells can operate as pacemakers
When stimulated by cAMP, they begin emitting regular pulses of cAMP

Spiral Waves
Persistence & propagation of spiral waves explained analytically (Tyson & al., 1989)
Rotate around a small core of of non-excitable cells
Propagate at higher frequency than circular
Therefore they dominate circular in collisions
But how do the spirals form initially?

Some Explanations
of Spiral Formation
Òthe origin of spiral waves remains obscureÓ (1997)
Traveling wave meets obstacle and is broken
Desynchronization of cells in their developmental path
Random pulse behind advancing wave front

Step 0: Passing Wave Front
Step 1: Random Excitation
Step 2: Beginning of Spiral
Step 3
Step 4
Step 5
Step 6: Rejoining & Reinitiation
Step 7: Beginning of New Spiral
Step 8
Formation of Double Spiral
StarLogo Simulation
Of Spiral Formation
Amoebas are immobile at timescale of wave movement
A fraction of patches are inert (grey)
A fraction of patches has initial concentration of cAMP
At each time step:
chemical diffuses
each patch responds to local concentration

Response of Patch
if patch is not refractory (brown) then
if local chemical > threshold then
set refractory period
produce pulse of chemical (red)
else
decrement refractory period
degrade chemical in local area

Demonstration of StarLogo Simulation of Spiral Formation
Run SlimeSpiral.slogo